Ventricular aneurysm - Biotech, Pharma and Life Science Channel
Ventricular aneurysms are usually aftereffects of myocardial infarctions. When the heart muscle (cardiac muscle) partially dies during a heart attack, a layer of muscle may survive, and being severely weakened, start to become an aneurysm. The blood may flow into the surrounding dead muscle and inflate the weakened flap of muscle into a bubble.
Ventricular aneurysms can be fatal. They are usually non-rupturing because they are lined by scar tissue.
Despite the fact that ventricular aneurysms usually grow at a very slow pace, they still pose a growing danger to wherever they are located in the heart. Usually, this type of aneurysm grows in the left ventricle. This bubble has the potential to block blood flow to the body, and thus limit the patient's stamina. In other cases, a similarly developed pseudoaneurysm "false aneurysm" may burst, and sometimes may cause the patient to decease. Also, blood clots may form on the inside of ventricular aneurysms, and form embolisms, which can cause ventricular failure or arrythmia, or a stroke. At this stage, treatment is a must.
Treatment is limited to surgery (ventricular reduction) for this defect of the heart. However, surgery is not required in most cases but, limiting the patient's physical activity levels to lower the risk of making the aneurysm bigger is advised.
Source; World News
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Companies
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Videos
OPCAB with ventricular aneurysm repair
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Medical and Biotech [MESH] Definitions
Defibrillators
Cardiac electrical stimulators that apply brief high-voltage electroshocks to the HEART. These stimulators are used to restore normal rhythm and contractile function in hearts of patients who are experiencing VENTRICULAR FIBRILLATION or ventricular tachycardia (TACHYCARDIA, VENTRICULAR) that is not accompanied by a palpable PULSE. Some defibrillators may also be used to correct certain noncritical dysrhythmias (called synchronized defibrillation or CARDIOVERSION), using relatively low-level discharges synchronized to the patient's ECG waveform. (UMDNS, 2003)
Andersen Syndrome
A form of inherited long QT syndrome (or LQT7) that is characterized by a triad of potassium-sensitive periodic paralysis, VENTRICULAR ECTOPIC BEATS, and abnormal features such as short stature, low-set ears, and SCOLIOSIS. It results from mutations of KCNJ2 gene which encodes a channel protein (INWARD RECTIFIER POTASSIUM CHANNELS) that regulates resting membrane potential.
Brugada Syndrome
An autosomal dominant defect of cardiac conduction that is characterized by an abnormal ST-segment in leads V1-V3 on the ELECTROCARDIOGRAM resembling a right BUNDLE-BRANCH BLOCK; high risk of VENTRICULAR TACHYCARDIA; or VENTRICULAR FIBRILLATION; SYNCOPAL EPISODE; and possible sudden death. This syndrome is linked to mutations of gene encoding the cardiac SODIUM CHANNEL alpha subunit.
Ventricular Septum
The muscular structure separating the right and the left lower chambers (HEART VENTRICLES) of the heart. The ventricular septum consists of a very small membranous portion just beneath the AORTIC VALVE, and a large thick muscular portion consisting of three sections including the inlet septum, the trabecular septum, and the outlet septum.
Ventricular Flutter
A potentially lethal cardiac arrhythmia characterized by an extremely rapid, hemodynamically unstable ventricular tachycardia (150-300 beats/min) with a large oscillating sine-wave appearance. If untreated, ventricular flutter typically progresses to VENTRICULAR FIBRILLATION.
