Recent Publications on : |  |
Modulation of nucleotide excision repair in human lymphocytes by genetic and dietary factors.
Gene-environment interactions determine inter-individual variations in...
Direct Link | 3rd November, 2009
| Department of Health Risk Analysis and Toxicology, Nutrition and
|
Nucleic acid binding activity of human Cockayne syndrome B protein and identification of Ca(2+) as a novel metal cofactor.
The Cockayne syndrome group B protein (CSB) is a member of the SWI/SNF2...
Direct Link | 15th October, 2009
| Laboratory of Molecular Gerontology, National Institute on Aging, National
|
| [Genetic aspects of age-related macular degeneration]
| 18th September, 2009
| Z Katedry Genetyki Molekularnej Uniwersytetu Lodzkiego.
|
Cockayne syndrome group B protein is engaged in processing of DNA adducts of lipid peroxidation product trans-4-hydroxy-2-nonenal.
Cockayne syndrome complementation group B (CSB) protein is engaged in...
Direct Link | 22nd August, 2009
| Institute of Biochemistry and Biophysics, Polish Academy of Sciences,
|
Laryngeal cancer risk associated with smoking and alcohol consumption is modified by genetic polymorphisms in ERCC5, ERCC6 and RAD23B but not by polymorphisms in five other nucleotide excision repair genes.
Laryngeal cancer is known to be associated with smoking and high alcohol...
Direct Link | 21st August, 2009
| Division of Epigenomics and Cancer Risk Factors, German Cancer Research
|
Elevated lung cancer risk is associated with deficiencies in cell cycle checkpoints: Genotype and phenotype analyses from a case-control study.
Cell cycle checkpoints play critical roles in the maintenance of genomic...
Direct Link | 25th July, 2009
| Cancer Genetics and Epidemiology Program, Lombardi Comprehensive Cancer
|
Congenital DNA repair deficiency results in protection against renal ischemia reperfusion injury in mice.
Cockayne syndrome and other segmental progerias with inborn defects in DNA...
Direct Link | 27th June, 2009
| Department of Surgery, Erasmus Medical Center, Rotterdam, The Netherlands.
|
Accumulation of mitochondrial DNA damage and bioenergetic dysfunction in CSB defective cells.
Cockayne syndrome (CS) is a complex, progressive disease that involves...
Direct Link | 24th June, 2009
| Institute of Clinical Biochemistry, Faculty division Rikshospitalet,
|
Prenatal diagnosis of Cockayne syndrome type A based on the identification of two novel mutations in the ERCC8 gene.
Back Cockayne syndrome (CS; MIM 133540-216400) is a rare autosomal...
Direct Link | 16th June, 2009
| Fondazione Policlinico
|
Cockayne syndrome group B protein stimulates repair of formamidopyrimidines by NEIL1 DNA glycosylase.
Cockayne syndrome (CS) is a premature aging condition characterized by...
Direct Link | 2nd June, 2009
| Laboratory of Molecular Gerontology, NIA Intramural Research Program,
|
8-Oxoguanine DNA glycosylase (Ogg1) causes a transcriptional inactivation of damaged DNA in the absence of functional Cockayne syndrome B (Csb) protein.
We have analysed the effect of oxidative guanine lesions on the expression...
Direct Link | 28th May, 2009
| Johannes Gutenberg University of Mainz, Institute of Pharmacy, Mainz,
|
| Cockayne syndrome B protein (CSB): linking p53, HIF-1 and p300 to robustness, lifespan, cancer and cell fate decisions.
| 29th April, 2009
| MRC Clinical Sciences Centre, Faculty of Medicine, Imperial College
|
Accumulation of (5'S)-8,5'-cyclo-2'-deoxyadenosine in organs of Cockayne syndrome complementation group B gene knockout mice.
Cockayne syndrome (CS) is a human genetic disorder characterized by...
Direct Link | 8th April, 2009
| Chemical Science and Technology Laboratory, National Institute of
|
Absence of ERCC6 gene mutation in common cancers of Korean patients.
Cockayne syndrome (CS) is a premature aging condition characterized by...
Direct Link | 20th February, 2009
|
|
8-Oxoguanine-mediated transcriptional mutagenesis causes Ras activation in mammalian cells.
8-Oxoguanine (8OG) is efficiently bypassed by RNA polymerases in vitro and...
Direct Link | 6th January, 2009
| Department of Biochemistry, Emory University School of Medicine, Atlanta,
|
