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NAIP
Gene Information from Publications |
| Publication Link |
Summary of findings |
| 18071605 |
Observational study of genotype prevalence. (HuGE Navigator) |
| 17923748 |
While there was no evidence of NAIP expression in the normal breast tissue, NAIP was expressed in all breast cancer samples. |
| 17903057 |
80% neuronal apoptosis inhibitory protein gene deletion in 5q-spinal muscular atrophy patients (91% spinal muscular atrophy-I, 50% spinal muscular atrophy-II and -III), and in 5% (two of forty) of spinal muscular atrophy parents, was found. |
| 17510375 |
a role for NAIP in increasing the survival of cells undergoing terminal differentiation as well as the possibility that the protein serves as an intestinal pathogen recognition protein was suggested |
| 15340105 |
Alterations in C/CAAT enhancer binding protein alpha and neuronal apoptosis inhibitory protein expression occurred in human adipose stromal-vascular cells after weight loss |
| 17222062 |
Multiple, domesticated long terminal repeats (LTRs) of endogenous retroviral elements provide NAIP promoter function in human, mouse, and rat. |
| 11912351 |
NAIP gene deletion was higher in type I spinal muscular atrophy than in type U or V. In type I patients lacking the NAIP gene, deterioration in their respiratory function is more rapid than in those type I patients retaining the NAIP gene. |
| 15280366 |
NAIP does not interact with Smac and requires ATP to bind caspase-9 |
| 12445469 |
NAIP:Structural requirements for binding hippocalcin and effects on survival of sympathetic neurons. |
| 12127562 |
NAIP-deltaEx10-11: a novel splice variant of the apoptosis inhibitor NAIP is differently expressed in drug-sensitive and multidrug-resistant HL60 leukemia cells. NAIP transcripts might be involved in tumor resistance to chemotherapeutic agents. | →View more information from publications. |
Recent Publications on NAIP: |  |
Diminished hippocalcin expression in Huntington's disease brain does not account for increased striatal neuron vulnerability as assessed in primary neurons.
Hippocalcin is a neuronal calcium sensor protein previously implicated in... | 23rd October, 2009
| Brain Mind Institute, Ecole Polytechnique Federale de Lausanne, Lausanne,
| J Neurochem. 2009 Oct;111(2):460-72. Epub 2009 Aug 17.
DOI Direct Link |
Transmigration across activated endothelium induces transcriptional changes, inhibits apoptosis, and decreases antimicrobial protein expression in human monocytes.
We investigated the hypothesis that transmigration drives monocyte... | 27th August, 2009
| *The Wallace H. Coulter Department of Biomedical Engineering, Georgia
| J Leukoc Biol. 2009 Aug 25.
DOI Direct Link |
Molecular analysis of the SMN and NAIP genes in Iranian spinal muscular atrophy patients.
BACKGROUND: Spinal muscular atrophy (SMA) is an autosomal recessive... | 26th August, 2009
| Department of Genetics, Faculty of Natural Sciences, University of Tabriz,
| Pediatr Int. 2009 Apr;51(2):193-6.
DOI Direct Link |
Evaluation of the SMN and NAIP genes in a family: homozygous deletion of the SMN2 gene in the fetus and outcome of the pregnancy.
BACKGROUND: Spinal muscular atrophy (SMA) is an autosomal recessive... | 25th July, 2009
| Department of Medical Genetics, Faculty of Medicine, Ege University,
| Genet Test Mol Biomarkers. 2009 Jun;13(3):287-8.
DOI Direct Link |
Phenotype modifiers of spinal muscular atrophy: the number of SMN2 gene copies, deletion in the NAIP gene and probably gender influence the course of the disease.
Spinal muscular atrophy (SMA) is an autosomal recessive neuromuscular... | 20th June, 2009
| Neuromuscular Unit, Mossakowski Medical Research Centre, Polish Academy of
| Acta Biochim Pol. 2009;56(1):103-8. Epub 2009 Mar 14.
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→View more research publications. |
NAIP results (if any) from reagent suppliers | 
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