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WAS
Gene Information from Publications |
| Publication Link |
Summary of findings |
| 18043243 |
The discovery of unique functional domains of Wiskott-Aldrich syndrome protein has been instrumental in defining mechanisms that control activation of Wiskott-Aldrich syndrome protein. Review. |
| 17890224 |
that WASP and WASP-interacting protein (WIP) form a complex at the phagocytic cup and that the WASP.WIP complex plays a critical role in the phagocytic cup formation. |
| 17703096 |
Chinese Wiskott-Aldrich Syndrome (WAS) patients had mutations involving exon 1 of the Wiskott-Aldrich syndrome protein (WASP) gene and none had the X-linked thrombocytopenia phenotype. |
| 17711847 |
the WIP-WASP complex plays an important role in WASP stabilization and NFAT activation |
| 17719003 |
Study provides a strategy that allows a strong suppression of WASp in CD34(+) cells and will facilitate future studies on the role of WASp in human cells. |
| 17400488 |
study found 28 novel WAS mutations in Wiskott-Aldrich syndrome and X-linked thrombocytopenia (7 missense, 1 nonsense, 1 nonstop change, 5 splice site mutations and 14 deletions or insertions) |
| 17724125 |
These findings reveal a novel mechanism for inhibition of myelopoiesis through defective mitosis and cytokinesis due to hyperactivation and mislocalization of actin polymerization. |
| 14559906 |
the interaction of the betaPIX.WASP.SPIN90 complex with Nck is crucial for stable cell adhesion and can be dynamically modulated by SPIN90 phosphorylation that is dependent on cell adhesion and ERK activation |
| 17296785 |
Thus, WASP appears to play an important role in the activation and suppressor function of nTreg cells, and a dysfunction or incorrect localization of nTreg cells may contribute to the development of autoimmunity in WAS patients. |
| 12591280 |
X-linked thrombocytopenia caused by a mutation in the WAS gene that disrupts interaction with the (WASP)-interacting protein (WIP). | →View more information from publications. |
Recent Publications on WAS: |  |
Modulation of inflammatory response by pentoxifylline is independent of heme oxygenase-1 pathway.
OBJECTIVE: It was reported that some effects of pentoxifylline (PTX) are... | 23rd October, 2009
| Department of Medical Biotechnology, Faculty of Biochemistry, Biophysics
| J Physiol Pharmacol. 2009 Jun;60(2):3-12.
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RADIal versus femoral approach for percutaneous coronary interventions in patients with Acute Myocardial Infarction (RADIAMI): A prospective, randomized, single-center clinical trial.
BACKGROUND: The transradial approach for percutaneous coronary... | 22nd October, 2009
| Silesian Center for Heart Diseases First Department of Cardiology, Medical
| Cardiol J. 2009;16(4):332-40.
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Zinc protoporphyrin IX, a heme oxygenase-1 inhibitor, demonstrates potent antitumor effects but is unable to potentiate antitumor effects of chemotherapeutics in mice.
BACKGROUND: HO-1 participates in the degradation of heme. Its products can... | 20th June, 2009
| Department of Immunology, Center of Biostructure Research, the Medical
| BMC Cancer. 2008 Jul 11;8:197.
DOI Direct Link |
Heme oxygenase-1 accelerates cutaneous wound healing in mice.
Heme oxygenase-1 (HO-1), a cytoprotective, pro-angiogenic and... | 6th June, 2009
| Department of Medical Biotechnology, Faculty of Biochemistry, Biophysics
| PLoS One. 2009 Jun 4;4(6):e5803.
DOI Direct Link |
The comparison of contrast echocardiography and tissue Doppler imaging for evaluation of reperfused myocardium in patients with acute anterior myocardial infarction.
BACKGROUND: Prediction of functional myocardial recovery post acute... | 8th April, 2009
| Silesian Centre for
| Cardiol J. 2008;15(6):548-54.
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→View more research publications. |
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