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ADAMTS1
Gene Information from Publications |
| Publication Link |
Summary of findings |
| 18192475 |
ADAMTS-1 was significantly upregulated in thrombosed hemodialysis grafts. |
| 18076023 |
Expression of several genes associated with angiogenesis was altered during transition into androgen-independency. Among these, a significant decrease was found for ADAMTS1, whose expression inversely correlated with microvessel density. |
| 18174457 |
Clinical trial of gene-disease association, gene-environment interaction, and pharmacogenomic / toxicogenomic. (HuGE Navigator) |
| 17560840 |
These results show that ADAMTS-1 has both the opportunity in bone and capability in vitro to induce collagen type I processing, together with a positive influence on osteoblastic three-dimensional growth. |
| 15967414 |
Expression of ADAMTS1 was downregulated by cAMP in normal, but not in SV40-transformed cells, suggesting that in normal cells epiregulin and amphiregulin activity is downregulated by a feedback mechanism that may be lost in SV40-transformed cells |
| 16641089 |
role of TFPI-2 as a maintenance factor of extracellular remodeling suggests the indirect function of ADAMTS1 as an additional homeostatic player by its ability to alter the extracellular location of TFPI-2 |
| 12716911 |
ADAMTS1 significantly blocks VEGFR2 phosphorylation due to direct binding and sequestration of VEGF165, with consequent suppression of endothelial cell proliferation. |
| 15878613 |
specific distribution pattern of ADAMTS-1 in a variety of organs during embryogenesis suggests a role of the molecule in tissue remodelling, vasculogenesis and angiogenesis |
| 14996435 |
ADAMTS-1 expression was significantly reduced in the presence of HDL subfraction 3. |
| 16314835 |
Overexpression of ADAMTS-1 promotes pulmonary metastasis of mammary carcinoma and Lewis lung carcinoma cells and that a proteinase-dead mutant of ADAMTS-1 inhibits their metastasis | →View more information from publications. |
Recent Publications on ADAMTS1: |  |
Role of endogenous angiogenesis inhibitors in Down syndrome.
New blood vessel growth via angiogenesis is a fundamental process in both... | 16th October, 2009
| Vascular Biology Program, Department of Surgery, Children's Hospital,
| J Craniofac Surg. 2009 Mar;20 Suppl 1:595-6.
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Positional identification of variants of Adamts16 linked to inherited hypertension.
A previously reported blood pressure (BP) quantitative trait locus on rat... | 15th October, 2009
| Physiological Genomics Laboratory, Department of Physiology and
| Hum Mol Genet. 2009 Aug 1;18(15):2825-38. Epub 2009 May 7.
DOI Direct Link |
Proteolytic processing causes extensive heterogeneity of tissue matrilin forms.
The matrilins are a family of multidomain extracellular matrix proteins... | 29th September, 2009
| Center for Biochemistry, Medical Faculty, University of Cologne, Cologne,
| J Biol Chem. 2009 Aug 7;284(32):21545-56. Epub 2009 Jun 16.
DOI Direct Link |
FOS proliferating network construction in early colorectal cancer (CRC) based on integrative significant function cluster and inferring analysis.
The aim is to setup single distinguished molecular network. We constructed... | 18th September, 2009
| Center for Biomedical Engineering, Beijing University of Posts and
| Cancer Invest. 2009 Oct;27(8):816-24.
DOI Direct Link |
Candidate therapeutic agents for hepatocellular cancer can be identified from phenotype-associated gene expression signatures.
BACKGROUND: The presence of vascular invasion in hepatocellular cancer... | 18th September, 2009
| Department of Internal Medicine, College of Medicine, Ohio State
| Cancer. 2009 Aug 15;115(16):3738-48.
DOI Direct Link |
→View more research publications. |
ADAMTS1 results (if any) from reagent suppliers | 
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