Recent Publications on Calcium-Calmodulin-Dependent Protein Kinase Type 4: |  |
TRPC6 channels promote dendritic growth via the CaMKIV-CREB pathway.
The canonical transient receptor potential channels (TRPCs) are...
Direct Link | 8th October, 2008
| Laboratory of Neural Signal Transduction, Institute of Neuroscience,
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Critical role of TRPC6 channels in the formation of excitatory synapses.
The transient receptor potential canonical (TRPC) channels are...
Direct Link | 20th September, 2008
| Laboratory of Neural Signal Transduction, Institute of Neuroscience,
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Genetic enhancement of trace fear memory and cingulate potentiation in mice overexpressing Ca2+/calmodulin-dependent protein kinase IV.
Long-term potentiation (LTP) is a key cellular model for studying...
Direct Link | 2nd July, 2008
| Department of Physiology, University of Toronto, 1 King's College Circle,
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Difference in physiogenomics between male and female infertility.
Infertility is an important condition in reproductive medicine. According...
Direct Link | 21st June, 2008
| Wiwanitkit House, Bangkhae, Bangkok, Thailand; Hainan Medical College,
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o-GlcNAc transferase is activated by CaMKIV-dependent phosphorylation under potassium chloride-induced depolarization in NG-108-15 cells.
Post-translational modification of cellular proteins by beta-o-linked...
Direct Link | 12th June, 2008
| Department of Life Science, Division of Molecular and Life Science, Pohang
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Roles of calcium-stimulated adenylyl cyclase and calmodulin-dependent protein kinase IV in the regulation of FMRP by group I metabotropic glutamate receptors.
The fragile X syndrome is caused by the lack of fragile X mental...
Direct Link | 21st May, 2008
| Department of Physiology, Faculty of Medicine, University of Toronto,
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Neuroprotection by neurotrophic factors and membrane depolarization is regulated by calmodulin kinase IV.
Neurotrophic factors promote motoneuron (MN) survival through increased...
Direct Link | 6th May, 2008
| Cell Signaling and Apoptosis Group, Departament de Ciencies Mediques
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Adeno-associated virus (AAV)-mediated suppression of Ca2+/calmodulin kinase IV activity in the nucleus accumbens modulates emotional behaviour in mice.
BACKGROUND: Calcium/calmodulin-dependent protein kinase IV (CaMKIV)...
Direct Link | 9th April, 2008
| Central Institute of Mental Health (ZI), Department of Psychopharmacology,
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Calmodulin-dependent kinase IV links Toll-like receptor 4 signaling with survival pathway of activated dendritic cells.
Microbial products, including lipopolysaccharide (LPS), an agonist of...
Direct Link | 12th March, 2008
| Department of Molecular and Cellular Biology and Pathology, Federico II
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| Physiological roles of the Ca2+/CaM-dependent protein kinase cascade in health and disease.
| 6th February, 2008
| Department of Pharmacology and Cancer Biology, Duke University Medical
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GATA-2 functions downstream of BMPs and CaM KIV in ectodermal cells during primitive hematopoiesis.
In Xenopus, primitive blood originates from the mesoderm, but extrinsic...
Direct Link | 4th January, 2008
| Department of Cell and Developmental Biology, Oregon Health and Science
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CaMKII and CaMKIV mediate distinct prosurvival signaling pathways in response to depolarization in neurons.
By fusing the CaMKII-inhibitory peptide AIP to GFP, we constructed a...
Direct Link | 7th December, 2007
| Department of Biological Sciences, University of Iowa, Iowa City, IA
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Nicotine prevents disruption of the late phase LTP-related molecular cascade in adult-onset hypothyroidism.
We have shown previously that chronic nicotine treatment reverses...
Direct Link | 1st November, 2007
| Department of Pharmacological and Pharmaceutical Sciences, College of
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| [Effects of calcium and calmodulin dependent kinase against hypoxic neuronal injury]
| 31st October, 2007
| Department of Pediatrics, West China Second University Hospital, Sichuan
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X-box-binding protein 1 activates lytic Epstein-Barr virus gene expression in combination with protein kinase D.
Epstein-Barr virus (EBV) establishes a latent form of infection in memory...
Direct Link | 15th September, 2007
| Department of Medicine, Lineberger Comprehensive Cancer Center, University
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