Recent Publications on Hemophilia B: |  |
Carbon monoxide releasing molecule-2 increases the velocity of thrombus growth and strength in hemophilia A, hemophilia B and factor VII-deficient plasmas.
Carbon monoxide derived from carbon monoxide releasing molecules (CORMs)...
Direct Link | 20th November, 2009
| aDepartments of Anesthesiology, USA bSurgery, The University of Alabama at
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Proteasome Inhibitors Decrease AAV2 Capsid derived Peptide Epitope Presentation on MHC Class I Following Transduction.
Adeno-associated viral (AAV) vectors are an extensively studied and highly...
Direct Link | 12th November, 2009
| Department of Pediatrics, Division of Hematology, The Children's Hospital
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Genotype analysis identifies the cause of the "royal disease".
The "royal disease," a blood disorder transmitted from Queen Victoria to...
Direct Link | 10th October, 2009
| University of Massachusetts Medical School, 303 Belmont Street, Worcester,
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| The Jeremiah Metzger Lecture: gene therapy for inherited disorders: from Christmas disease to Leber's amaurosis.
| 22nd September, 2009
| 3615 Civic Center Boulevard, 302D Abramson Pediatric Research Center,
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| Therapeutic properties and safety of recombinant factor VIII and factor IX.
| 18th September, 2009
| Department of Hematology, Jagiellonian University School of Medicine,
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| Novel human pathological mutations. Gene symbol: F9. Disease: Haemophilia B.
| 9th September, 2009
| Medical Genetics, Centre of Excellence in Molecular Biology, Lahore,
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| [Hemorrhagic parapneumonic effusion in a 64 year-old patient as the first symptom of hemophilia B]
| 9th September, 2009
| Katedra i Klinika Chorob Wewnetrznych, Pneumonologii i Alergologii
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Spectrum of inherited bleeding disorders in southern Iran, before and after the establishment of comprehensive coagulation laboratory.
The objective of the present study was to determine the pattern of...
Direct Link | 28th August, 2009
| Hematology Research Center, Shiraz University of Medical Sciences, Shiraz,
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Hemophilia B is a quasi-quantitative condition with certain mutations showing phenotypic plasticity.
Hemophilia B has been considered as a classical monogenic disorder with...
Direct Link | 25th August, 2009
| Functional Genomics Unit, Institute of Genomics and Integrative Biology,
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| [Trends in development of self-complementary adeno-associated virus vector]
| 13th August, 2009
| Institute of Molecular Medicine, Huaqiao University, Fujian 362021, China.
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Impact of the underlying mutation and the route of vector administration on immune responses to factor IX in gene therapy for hemophilia B.
Immune responses to factor IX (F.IX), a major concern in gene therapy for...
Direct Link | 16th July, 2009
| Department of Pediatrics, University of Florida, Gainesville, Florida
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Abnormal hemostasis in a knock-in mouse carrying a variant of Factor IX with impaired binding to collagen type IV.
Summary Background: FIX binds to collagen type IV but this binding has no...
Direct Link | 9th July, 2009
| Department of Biology, University of North Carolina at Chapel Hill, Chapel
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Prophylactic immune tolerance induced by changing the ratio of antigen-specific effector to regulatory T cells.
BACKGROUND: Gene and protein replacement therapies for inherited protein...
Direct Link | 9th July, 2009
| Department of Pediatrics, University of Florida, Gainesville, FL 32610,
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Changes in a hemophilic pseudotumor of the mandible that developed over a 20-year period.
BACKGROUND: Gene and protein replacement therapies for inherited protein...
Direct Link | 7th July, 2009
| Department of Oral and Maxillofacial Surgery, Dental Hospital, East-West
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