Recent Publications on Map Kinase Kinase 4: |  |
Lipopolysaccharide-evoked activation of p38 and JNK leads to an increase in ICAM-1 expression in Schwann cells of sciatic nerves.
Lipopolysaccharide is a major constituent of the outer membrane of...
Direct Link | 4th October, 2008
| Jiangsu Province Key Laboratory of Neuroregeneration, Nantong University,
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Signaling pathways required for macrophage scavenger receptor-mediated phagocytosis: analysis by scanning cytometry.
BACKGROUND: Scavenger receptors are important components of the innate...
Direct Link | 1st October, 2008
| Harvard School of Public Health, Molecular and Integrative Physiological
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Lipopolysaccharide (LPS) of Porphyromonas gingivalis induces IL-1beta, TNF-alpha and IL-6 production by THP-1 cells in a way different from that of Escherichia coli LPS.
Lipopolysaccharide (LPS) derived from the periodontal pathogen...
Direct Link | 27th September, 2008
| Department of Stomatology, The Second Afiliated Hospital, School of
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| The association of deamidation of Bcl-xL and translocation of Bax to the mitochondria through activation of JNK in the induction of apoptosis by treatment with GSH-conjugated DXR.
| 24th September, 2008
| Department of Biochemistry, Jikei University School of Medicine,
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Induction of apoptosis in human cancer cells by candidaspongiolide, a novel sponge polyketide.
BACKGROUND: Candidaspongiolide (CAN), a novel polyketide from a marine...
Direct Link | 19th September, 2008
| Tumor Hypoxia Laboratory, SAIC-Frederick, Inc., National Cancer Institute,
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A TLR4/MD2 fusion protein inhibits LPS-induced pro-inflammatory signaling in hepatic stellate cells.
Activated hepatic stellate cells (HSCs) play a key role in hepatic...
Direct Link | 19th September, 2008
| Department of Medicine, University of California San Diego, 9500 Gilman
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| [Redox-dependent regulation of apoptosis as a function of intracellular reactive oxygen species in oxidative stress]
| 16th September, 2008
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| Synoviocyte innate immune responses: I. Differential regulation of interferon responses and the JNK pathway by MAPK kinases.
| 16th September, 2008
| Division of Rheumatology, Allergy and Immunology, University of
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Regulatory mechanism of TNFalpha autoregulation in HaCaT cells: the role of the transcription factor EGR-1.
Tumor necrosis factor alpha (TNFalpha) is a pro-inflammatory cytokine...
Direct Link | 11th September, 2008
| Department of Dermatology, Korea University College of Medicine, Seoul
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XIAP regulates intracellular ROS by enhancing antioxidant gene expression.
XIAP (X chromosome-linked inhibitor of apoptosis) is a member of the...
Direct Link | 10th September, 2008
| Department of Vascular Biology and Thrombosis Research, Medical University
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Role of oxidant stress on AT1 receptor expression in neurons of rabbits with heart failure and in cultured neurons.
We have previously reported that the expression of Angiotensin II (Ang II)...
Direct Link | 9th August, 2008
| Department of Cellular and Integrative Physiology, University of Nebraska
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Chronic insulin treatment causes insulin resistance in 3T3-L1 adipocytes through oxidative stress.
Insulin resistance and hyperinsulinemia are commonly present in obesity...
Direct Link | 8th August, 2008
| Key Laboratory of Nutrition and Metabolism, Institute for Nutritional
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Chromium (VI) induces insulin resistance in 3T3-L1 adipocytes through elevated reactive oxygen species generation.
Reactive oxygen species (ROS) have been proposed to be involved in the...
Direct Link | 8th August, 2008
| Key Laboratory of Nutrition and Metabolism, Institute for Nutritional
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25-hydroxycholesterol induces mitochondria-dependent apoptosis via activation of glycogen synthase kinase-3beta in PC12 cells.
25-hydroxycholesterol (25-OH-chol) induces apoptosis in many cell types....
Direct Link | 8th August, 2008
| College of Pharmacy, Seoul National University, Seoul, Republic of Korea.
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Important genetic checkpoints for insulin resistance in salt-sensitive (S) Dahl rats.
Despite the marked advances in research on insulin resistance (IR) in...
Direct Link | 29th July, 2008
| Department of Cellular and Molecular Medicine, University of Ottawa Heart
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