«Proto-Oncogene Proteins C-Raf

Below is an extended Proto-Oncogene Proteins C-Raf research listing. Click here to return to the Proto-Oncogene Proteins C-Raf main in depth entry.

Recent Publications on Proto-Oncogene Proteins C-Raf:
Development of a yeast protein fragment complementation assay (PCA) system using dihydrofolate reductase (DHFR) with specific additives. A yeast protein fragment complementation assay (PCA) system based on... Direct Link		15th November, 2008	Department of Pharmacy, School of Pharmacy, Hyogo University of Health
Knockdown of B-Raf impairs spindle formation and the mitotic checkpoint in human somatic cells.		4th November, 2008	Molecular Oncology Program, H. Lee Moffitt Cancer Center & Research
Hsp90-dependent activation of protein kinases is regulated by chaperone-targeted dephosphorylation of Cdc37. Activation of protein kinase clients by the Hsp90 system is mediated by... Direct Link		31st October, 2008	Section of Structural Biology, Institute of Cancer Research, Chester
CRAF autophosphorylation of serine 621 is required to prevent its proteasome-mediated degradation. The CRAF protein kinase regulates proliferative, differentiation, and... Direct Link		31st October, 2008	Department of Biochemistry, Henry Wellcome Building, University of
The resveratrol analogue 3,5,3',4',5'-pentahydroxy-trans-stilbene inhibits cell transformation via MEK. Resveratrol, present in grapes and red wine, is reported to be a natural... Direct Link		23rd October, 2008	Hormel Institute, University of Minnesota, Austin, MN 55912, USA.
Characterization of the topology and functional domains of RKTG. RKTG (Raf kinase trapping to Golgi) is exclusively localized at the Golgi... Direct Link		22nd October, 2008	Key Laboratory of Nutrition and Metabolism, Institute for Nutritional
Dual targeting of HSC70 and HSP72 inhibits HSP90 function and induces tumor-specific apoptosis. Heat-shock protein 70 (HSP70) isoforms contribute to tumorigenesis through... Direct Link		16th October, 2008	Signal Transduction and Molecular Pharmacology Team, Cancer Research UK
Epigenetic silencing of cell adhesion molecule 1 in different cancer progenitor cells of transgenic c-Myc and c-Raf mouse lung tumors. Understanding molecular mechanisms underlying lung cancer is a... Direct Link		15th October, 2008	Molecular Medicine and Medical Biotechnology, Fraunhofer Institute of
Sensitization to the lysosomal cell death pathway by oncogene-induced down-regulation of lysosome-associated membrane proteins 1 and 2. Expression and activity of lysosomal cysteine cathepsins correlate with... Direct Link		3rd October, 2008	Apoptosis Department and Centre for Genotoxic Stress Response, Institute
K-ras nanoclustering is subverted by overexpression of the scaffold protein galectin-3. The spatial organization of K-Ras proteins into nanoclusters on the plasma... Direct Link		3rd October, 2008	Department of Neurobiology, The George S. Wise Faculty of Life Sciences,
HDAC6 inhibition enhances 17-AAG--mediated abrogation of hsp90 chaperone function in human leukemia cells. Histone deacetylase 6 (HDAC6) is a heat shock protein 90 (hsp90)... Direct Link		1st October, 2008	Medical College of Georgia (MCG) Cancer Center, Augusta, GA 30912, USA.
Cross-talk between aryl hydrocarbon receptor and mitogen-activated protein kinase signaling pathway in liver cancer through c-raf transcriptional regulation. c-raf is a serine-threonine kinase and a downstream effector of ras... Direct Link		27th September, 2008	Fraunhofer Institute of Toxicology and Experimental Medicine,
Activation of protein kinase Calpha signaling prevents cytotoxicity and mutagenicity following lead acetate in CL3 human lung cancer cells. Protein kinase C (PKC) family of serine/threonine protein kinases is... Direct Link		13th September, 2008	Molecular Carcinogenesis Laboratory, Institute of Biotechnology &
Endothelial nitric oxide synthase regulates N-Ras activation on the Golgi complex of antigen-stimulated T cells. Ras/ERK signaling plays an important role in T cell activation and... Direct Link		30th August, 2008	Departamento de Biologia Vascular e Inflamacion, Centro Nacional de
Elevated CRAF as a potential mechanism of acquired resistance to BRAF inhibition in melanoma. Activating BRAF kinase mutations arise in approximately 7% of all human... Direct Link		19th August, 2008	Center for Molecular Therapeutics, Massachusetts General Hospital Cancer

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