CaMKII-dependent phosphorylation of GluK5 mediates plasticity of kainate receptors
13:17 EST 4 Jan 2013 | Nature Publishing
CamKII governs long-term depression of kainate receptor (KAR) synaptic currents by phosphorylating the subunit GluK5, increasing its mobility and diffusion away from synaptic sites.
Original Article: CaMKII-dependent phosphorylation of GluK5 mediates plasticity of kainate receptors
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