Tachycardia-Induced Cardiomyopathy After Functional Closure of the Foramen Ovale in a Fetus.
Summary of "Tachycardia-Induced Cardiomyopathy After Functional Closure of the Foramen Ovale in a Fetus."
Although fetal tachyarrhythmias are relatively rare, they are an important causes of fetal morbidity and mortality. We report a 30-week pregnant woman with fetal tachycardia, fetal hydrops and ascites. Fetal heart rate was 230 bpm. Fetal heart rate was 230 bpm. M-mode echocardiography revealed a tachycardia with a 1:1 relationship between atrial and ventricular conduction and a short time interval between ventricular and atrial contraction. These findings suggested a diagnosis of atrioventricular re-entrant tachycardia. On the third day digoxin treatment, the fetal heart rate was still 225 bpm with a 1:1 relationship between atrial and ventricular conduction. At this time, no flow across the foramen ovale was detected. Functional closure of the foramen ovale was suspected.
Affiliation
Division of Pediatric Cardiology, Medical University of Vienna, Vienna, Austria, Elisabeth.Mlczoch@meduniwien.ac.at.
Journal Details
This article was published in the following journal.
Name: Pediatric cardiology
ISSN: 1432-1971
Pages:
Links
- PubMed Source: http://www.ncbi.nlm.nih.gov/pubmed/21350927
- DOI: http://dx.doi.org/10.1007/s00246-011-9926-7
Medical and Biotech [MESH] Definitions
Foramen Ovale, Patent
A condition in which the FORAMEN OVALE in the ATRIAL SEPTUM fails to close shortly after birth. This results in abnormal communications between the two upper chambers of the heart. An isolated patent ovale foramen without other structural heart defects is usually of no hemodynamic significance.
Foramen Ovale
An opening in the wall between the right and the left upper chambers (HEART ATRIA) of a fetal heart. Oval foramen normally closes soon after birth; when it fails to close the condition is called PATENT OVAL FORAMEN.
Persistent Fetal Circulation Syndrome
A syndrome of persistent PULMONARY HYPERTENSION in the newborn infant (INFANT, NEWBORN) without demonstrable HEART DISEASES. This neonatal condition can be caused by severe pulmonary vasoconstriction (reactive type), hypertrophy of pulmonary arterial muscle (hypertrophic type), or abnormally developed pulmonary arterioles (hypoplastic type). The newborn patient exhibits CYANOSIS and ACIDOSIS due to the persistence of fetal circulatory pattern of right-to-left shunting of blood through a patent ductus arteriosus (DUCTUS ARTERIOSUS, PATENT) and at times a patent foramen ovale (FORAMEN OVALE, PATENT).
Cardiomyopathies
A group of diseases in which the dominant feature is the involvement of the CARDIAC MUSCLE itself. Cardiomyopathies are classified according to their predominant pathophysiological features (DILATED CARDIOMYOPATHY; HYPERTROPHIC CARDIOMYOPATHY; RESTRICTIVE CARDIOMYOPATHY) or their etiological/pathological factors (CARDIOMYOPATHY, ALCOHOLIC; ENDOCARDIAL FIBROELASTOSIS).
Tachycardia, Supraventricular
A generic expression for any tachycardia that originates above the BUNDLE OF HIS.
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