Tuning of n- and p-Type Reduced Graphene Oxide Transistors with the Same Molecular Backbone.
Summary of "Tuning of n- and p-Type Reduced Graphene Oxide Transistors with the Same Molecular Backbone."
Play the field: A field-effect transistor using reduced graphene oxides was prepared through the reduction of graphene oxides connected between two electrodes. The use of pyrene with an electron-withdrawing group and an electron-donating group showed the p- and n-doping effects, respectively, while the pyrene backbone with no electron-withdrawing or -donating groups showed no doping effect.
Affiliation
NCRI, Center for Smart Molecular Memory, Department of Chemistry, Sungkyunkwan University, Suwon 440-746 (Republic of Korea), Fax: (+82) 31-299-5934.
Journal Details
This article was published in the following journal.
Name: Chemistry (Weinheim an der Bergstrasse, Germany)
ISSN: 1521-3765
Pages: 5155-9
Links
- PubMed Source: http://www.ncbi.nlm.nih.gov/pubmed/22434768
- DOI: http://dx.doi.org/10.1002/chem.201103554
Medical and Biotech [MESH] Definitions
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A CALCIUM-dependent, constitutively-expressed form of nitric oxide synthase found primarily in ENDOTHELIAL CELLS.
Nitric Oxide Synthase Type I
A CALCIUM-dependent, constitutively-expressed form of nitric oxide synthase found primarily in NERVE TISSUE.
Nitric Oxide Synthase Type Ii
A CALCIUM-independent subtype of nitric oxide synthase that may play a role in immune function. It is an inducible enzyme whose expression is transcriptionally regulated by a variety of CYTOKINES.
Hyperlipoproteinemia Type V
A severe type of hyperlipidemia, sometimes familial, that it is characterized by the elevation of both plasma CHYLOMICRONS and TRIGLYCERIDES contained in VERY-LOW-DENSITY LIPOPROTEINS. Type V hyperlipoproteinemia is often associated with DIABETES MELLITUS and is not caused by reduced LIPOPROTEIN LIPASE activity as in HYPERLIPOPROTEINEMIA TYPE I .
Nitric Oxide
A free radical gas produced endogenously by a variety of mammalian cells, synthesized from ARGININE by NITRIC OXIDE SYNTHASE. Nitric oxide is one of the ENDOTHELIUM-DEPENDENT RELAXING FACTORS released by the vascular endothelium and mediates VASODILATION. It also inhibits platelet aggregation, induces disaggregation of aggregated platelets, and inhibits platelet adhesion to the vascular endothelium. Nitric oxide activates cytosolic GUANYLATE CYCLASE and thus elevates intracellular levels of CYCLIC GMP.
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