Venous Thrombosis in Turner Syndrome

2014-08-27 03:33:42 | BioPortfolio


In the literature, cases of thrombosis in the venous system have been described in girls and women with Turner syndrome. By screening a group of women with Turner syndrome, the researchers wish to find out if this condition is more frequent in this patient population.


In the literature cases of thrombosis in the venous system has been described in girls and women with Turner syndrome. By screening a group of women with Turner syndrome we wish to find out if this condition is more frequent in this patient population. Participants are examined with screening blood tests evaluating thromboembolic disturbances.

Study Design

Observational Model: Case-Only, Time Perspective: Cross-Sectional


Thromboembolic Disease


Medical Department M (Endocrinology and Diabetes), Aarhus University Hospital
Aarhus C


Active, not recruiting


University of Aarhus

Results (where available)

View Results


Published on BioPortfolio: 2014-08-27T03:33:42-0400

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Stable blood coagulation factor activated by contact with the subendothelial surface of an injured vessel. Along with prekallikrein, it serves as the contact factor that initiates the intrinsic pathway of blood coagulation. Kallikrein activates factor XII to XIIa. Deficiency of factor XII, also called the Hageman trait, leads to increased incidence of thromboembolic disease. Mutations in the gene for factor XII that appear to increase factor XII amidolytic activity are associated with HEREDITARY ANGIOEDEMA TYPE III.

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