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Pramel7 mediates ground-state pluripotency through proteasomal–epigenetic combined pathways

11:09 EDT 29 Jun 2017 | Nature Publishing

Graf et al. demonstrate that Pramel7 maintains ground-state pluripotency by repressing DNA methylation through proteasomal degradation of UHRF1, thus linking the proteasome and epigenetics with cell fate regulation.

Original Article: Pramel7 mediates ground-state pluripotency through proteasomal–epigenetic combined pathways

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Bioinformatics is the application of computer software and hardware to the management of biological data to create useful information. Computers are used to gather, store, analyze and integrate biological and genetic information which can then be applied...