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Cancer Cells Exploit Double Bind in Immune Escape Act

18:30 EST 31 Jan 2020 | Genetic Engineering News

A mechanism that cancer cells exploit to escape T cells involves a mechanism involving two T-cell proteins: PD-1 and SHP2. Full activation of SHP2, a new report indicates, depends on SHP2 binding to PD-1 in two specific places. This finding suggests opportunities for the development of novel cancer immunotherapies, including small molecule approaches.

The post Cancer Cells Exploit Double Bind in Immune Escape Act appeared first on GEN - Genetic Engineering and Biotechnology News.

Original Article: Cancer Cells Exploit Double Bind in Immune Escape Act

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