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A mechanism that cancer cells exploit to escape T cells involves a mechanism involving two T-cell proteins: PD-1 and SHP2. Full activation of SHP2, a new report indicates, depends on SHP2 binding to PD-1 in two specific places. This finding suggests opportunities for the development of novel cancer immunotherapies, including small molecule approaches.
The post Cancer Cells Exploit Double Bind in Immune Escape Act appeared first on GEN - Genetic Engineering and Biotechnology News.
Original Article: Cancer Cells Exploit Double Bind in Immune Escape ActNEXT ARTICLE
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Cancer is not just one disease but many diseases. There are more than 100 different types of cancer. Most cancers are named for the organ or type of cell in which they start - for example, cancer that begins in the colon is called colon cancer; cancer th...