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Onset, time course and prediction of spasticity after stroke or traumatic brain injury.

08:00 EDT 10th May 2018 | BioPortfolio

Summary of "Onset, time course and prediction of spasticity after stroke or traumatic brain injury."

To describe spasticity from the onset of acquired brain injury, time course over the first year and factors associated with prediction of the development of spasticity
METHODS:
Recent relevant literature known to the authors, along with a complementary search yielding a total of 9 articles, represented the base for this scoping review.

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Journal Details

This article was published in the following journal.

Name: Annals of physical and rehabilitation medicine
ISSN: 1877-0665
Pages:

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PubMed Articles [15794 Associated PubMed Articles listed on BioPortfolio]

Warm-needle moxibustion for spasticity after stroke: A systematic review of randomized controlled trials.

Spasticity is a common post-stroke complication, and it results in substantial deterioration in the quality of life of patients. Although potential positive effects of warm-needle moxibustion on spast...

Wake-Up Stroke versus Stroke with Known Onset Time: Clinical and Multimodality CT Imaging Characteristics.

Current guidelines for the treatment of acute ischemic stroke are mainly based on the time between symptom onset and initiation of treatment. This time is unknown in patients with wake-up stroke (WUS)...

Post-stroke spasticity and shoulder pain prevalence decrease over the last 15 years.

The usual complications after recent stroke such as disabling spasticity and shoulder pain seemed less frequent in recent years. This study examined the frequency of spasticity and shoulder pain in re...

Introduction of direct oral anticoagulant within 7 days of stroke onset: a nomogram to predict the probability of 3-month modified Rankin Scale score > 2.

In clinical practice, direct oral anticoagulants (DOACs) are often started earlier (≤ 7 days) than in randomized clinical trials after stroke. We aimed to develop a nomogram model incorporating t...

Cryotherapy reduces muscle spasticity but does not affect proprioception in ischemic stroke: a randomized sham-controlled crossover study.

To evaluate the immediate effects of cryotherapy (using an ice pack) on ankle joint position sense (JPS) and the degree of spasticity following chronic hemiparetic stroke.

Clinical Trials [7397 Associated Clinical Trials listed on BioPortfolio]

Asian Botulinum Clinical Trial Designed for Early Stroke Spasticity

The aim of this clinical study is to investigate the efficacy and safety of Dysport® in patients with early onset of upper limb spasticity within 2-12 weeks after stroke.

Arm Spasticity - Non-Interventional Study Early BIRD (BoNT Treatment: Initial and Repeated Documentation) [AS-NIS Early BIRD]

Botulinum toxin A (BoNT-A) is effective and safe in alleviating post-stroke spasticity and reducing the burden of associated symptoms. The hypothesis for this non-interventional study in ...

Predictors Factors and Time of Onset of Spasticity and Their Relationship With the Functionality and Quality of Life.

Spasticity is part of the syndrome motor neuron following a Stroke. It is a clinical sign of great complexity that generates disability, limiting the function and altering the quality of l...

Sensorimotor Changes in Stroke Following Mindfulness

Roughly 30% of stroke survivors experience spasticity, a velocity-dependent increase in stretch reflexes. In this pilot study, the investigators aimed to examine the effects of mindfulness...

Efficacy and Safety Study of Botulinum Toxin Type A Against Placebo to Treat Spasticity in the Leg After a Stroke

The purpose of this study is to determine whether injections of Botulinum toxin type A into muscles of the leg are effective in treating patients with increased muscle tension/uncontrollab...

Medical and Biotech [MESH] Definitions

A class of traumatic stress disorders with symptoms that last more than one month. There are various forms of post-traumatic stress disorder, depending on the time of onset and the duration of these stress symptoms. In the acute form, the duration of the symptoms is between 1 to 3 months. In the chronic form, symptoms last more than 3 months. With delayed onset, symptoms develop more than 6 months after the traumatic event.

Anxiety disorders manifested by the development of characteristic symptoms following a psychologically traumatic event that is outside the normal range of usual human experience. Symptoms include re-experiencing the traumatic event, increased arousal, and numbing of responsiveness to or reduced involvement with the external world. Traumatic stress disorders can be further classified by the time of onset and the duration of these symptoms.

Predicting the time of OVULATION can be achieved by measuring the preovulatory elevation of ESTRADIOL; LUTEINIZING HORMONE or other hormones in BLOOD or URINE. Accuracy of ovulation prediction depends on the completeness of the hormone profiles, and the ability to determine the preovulatory LH peak.

The time from the onset of a stimulus until a response is observed.

The interval of time between onset of symptoms and receiving therapy.

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