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Priming of Pop-out does not provide reliable measures of target activation and distractor inhibition in selective attention: Evidence from a large-scale online study.

08:00 EDT 6th June 2018 | BioPortfolio

Summary of "Priming of Pop-out does not provide reliable measures of target activation and distractor inhibition in selective attention: Evidence from a large-scale online study."

Lamy, Antebi, Aviani, and Carmel (2008) reported that in a simple search task where participants located an odd coloured circle, the inter-trial relations could be used to derive robust and independent measures of target activation and distractor inhibition. When a target feature repeated there was a benefit, and when the previous target feature became the distractor feature there was a cost. These two measures correlated and were taken to reflect a measure of target activation. When the distractor feature repeated there was a benefit and when the previous distractor feature became the current target feature there was a cost, these two measures correlated and were taken to reflect a measure of distractor inhibition. In the current study we examined the same colour search task online on a large group of 312 participants. The results revealed significant effects of target and distractor repetition and switching. However, the correlations reported by Lamy et al. (2008) were non-significant. Instead we found the correlations between the two measures of repetition and the two measures of switching.

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Name: Vision research
ISSN: 1878-5646
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Medical and Biotech [MESH] Definitions

Class I-restricted activation of CD8-POSITIVE LYMPHOCYTES resulting from ANTIGEN PRESENTATION of exogenous ANTIGENS (cross-presentation). This is in contrast to normal activation of these lymphocytes (direct-priming) which results from presentation of endogenous antigens.

A product of COMPLEMENT ACTIVATION cascade, regardless of the pathways, that forms transmembrane channels causing disruption of the target CELL MEMBRANE and cell lysis. It is formed by the sequential assembly of terminal complement components (COMPLEMENT C5B; COMPLEMENT C6; COMPLEMENT C7; COMPLEMENT C8; and COMPLEMENT C9) into the target membrane. The resultant C5b-8-poly-C9 is the "membrane attack complex" or MAC.

A conserved AMINO ACID SEQUENCE located in the intracellular domains of a family of transmembrane proteins involved in various IMMUNE RESPONSES. The CONSENSUS SEQUENCE of this motif is YXXL(or I)X(6-8)YXXL(or I) (where X denotes any amino acid). When phosphorylated ITAM motifs provide docking sites for PROTEIN TYROSINE KINASES of the Syk family thus forming signaling complexes which lead to activation of immune responses.

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The sequential activation of serum COMPLEMENT PROTEINS to create the COMPLEMENT MEMBRANE ATTACK COMPLEX. Factors initiating complement activation include ANTIGEN-ANTIBODY COMPLEXES, microbial ANTIGENS, or cell surface POLYSACCHARIDES.

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