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Air pollution: A silent common killer for stroke and dementia.

07:00 EST 1st January 2018 | BioPortfolio

Summary of "Air pollution: A silent common killer for stroke and dementia."

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This article was published in the following journal.

Name: International journal of stroke : official journal of the International Stroke Society
ISSN: 1747-4949
Pages: 1747493018784476

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Dementia and Atrial Fibrillation: Pathophysiological Mechanisms and Therapeutic Implications.

Atrial fibrillation increases the risk of stroke by a factor of 4- to 5-fold, and dementia is a common consequence of stroke. However, atrial fibrillation has been associated with cognitive impairment...

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Previous studies have shown that patients with dementia receive less testing and treatment for stroke.

Plasma β-Amyloids and Tau Proteins in Patients with Vascular Cognitive Impairment.

Increases in plasma of β-amyloids (Aβ) and tau proteins have been noted in patients with Alzheimer's dementia (AD). Our study investigated the associations of plasma Aβ and tau proteins with dement...

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Longitudinal studies are needed to understand the long-term associations between stroke and dementia.

Association between ambient air pollution and daily hospital admissions for ischemic stroke: A nationwide time-series analysis.

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Clinical Trials [3941 Associated Clinical Trials listed on BioPortfolio]

Cutaneous Silent Period and Spasticity

The cutaneous silent period (CSP) is a brief transient suppression of the voluntary muscle contraction that follows a noxious cutaneous nerve stimulation. Studies in patients with central ...

Stepwise Screening for Silent Atrial Fibrillation After Stroke

The main hypothesis of this work is that an approach combining clinical parameters and biomarker assays could improve the understanding and prediction of the occurrence of silent atrial fi...

Risk of Stroke in Pulmonary Embolism With a Patent Foramen Ovale (PFO)

Pulmonary embolism is associated with a small but definite risk of paradoxical embolism in patients with a patent foramen ovale (PFO). While neurologic complications are unfrequent the inc...

Silent Atrial Fibrillation - Screening of High-risk Groups for Atrial Fibrillation (The Silence Study)

The primary aim of the present study is to screen high-risk type 2 diabetes patients and heart failure patients without a history of atrial fibrillation (AF) or ongoing oral anticoagulatio...

Evaluating the Effects of Hormone Replacement Therapy on Brain Function (The WHIMS-MRI Study)

A silent stroke is a type of stroke that does not produce any severe symptoms, but may slightly affect memory or thinking abilities. Women who take hormone replacement therapy (HRT) may ha...

Medical and Biotech [MESH] Definitions

Restoration of functions to the maximum degree possible in a person or persons suffering from a stroke.

A familial disorder inherited as an autosomal dominant trait and characterized by the onset of progressive CHOREA and DEMENTIA in the fourth or fifth decade of life. Common initial manifestations include paranoia; poor impulse control; DEPRESSION; HALLUCINATIONS; and DELUSIONS. Eventually intellectual impairment; loss of fine motor control; ATHETOSIS; and diffuse chorea involving axial and limb musculature develops, leading to a vegetative state within 10-15 years of disease onset. The juvenile variant has a more fulminant course including SEIZURES; ATAXIA; dementia; and chorea. (From Adams et al., Principles of Neurology, 6th ed, pp1060-4)

Stroke caused by lacunar infarction or other small vessel diseases of the brain. It features hemiparesis (see PARESIS), hemisensory, or hemisensory motor loss.

A set of nuclear proteins in SACCHAROMYCES CEREVISIAE that are required for the transcriptional repression of the silent mating type loci. They mediate the formation of silenced CHROMATIN and repress both transcription and recombination at other loci as well. They are comprised of 4 non-homologous, interacting proteins, Sir1p, Sir2p, Sir3p, and Sir4p. Sir2p, an NAD-dependent HISTONE DEACETYLASE, is the founding member of the family of SIRTUINS.

Heterogeneous group of neurodegenerative disorders characterized by frontal and temporal lobe atrophy associated with neuronal loss, gliosis, and dementia. Patients exhibit progressive changes in social, behavioral, and/or language function. Multiple subtypes or forms are recognized based on presence or absence of TAU PROTEIN inclusions. FTLD includes three clinical syndromes: FRONTOTEMPORAL DEMENTIA, semantic dementia, and PRIMARY PROGRESSIVE NONFLUENT APHASIA.

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