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Synchronization plays a fundamental role in healthy cognitive and motor function. Two issues are still poorly understood. First, how synchronization depends on the interplay between local dynamics, coupling and topology? Second, how prone to synchronization a network is, given its topological organization? To investigate the synchronizability of both anatomical and functional brain networks various studies resorted to the Master Stability Function (MSF) formalism, an elegant tool which allows analysing the stability of synchronous states in a dynamical system consisting of many coupled oscillators. Here, we argue that brain dynamics does not fulfil the formal criteria under which synchronizability is usually quantified and, perhaps more importantly, this measure refers to a global dynamical condition that never holds in the brain (not even in the most pathological conditions), and therefore no neurophysiological conclusions should be drawn based on it. We discuss the meaning of synchronizability and its applicability to neuroscience and propose alternative ways to quantify brain networks synchronization.
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At present, differential diagnosis of MRI enhancing lesions can still be challenging. Preoperative imaging is a valuable tool characterized by high informative value, even if false-positive and false-...
Humans exhibit a remarkable capacity for cooperation among genetically unrelated individuals. Yet, human cooperation is neither universal, nor stable. Instead, cooperation is often bounded to members ...
Sleep facilitates the extraction of semantic regularities amongst newly encoded memories, which may also lead to increased false memories. We investigated sleep stage proportions and sleep spindles in...
One-sided t-tests are widely used in neuroimaging data analysis. While such a test may be applicable when investigating specific regions and prior information about directionality is present, we argue...
Imagination is an internally-generated process, where one can make oneself or other people appear as protagonists of a scene. How does the brain tag the protagonist of an imagined scene, as being ones...
The overall objective is to examine if participating in an SMS-transmitted health educational program targeting a cluster of dietary factors over a period of weeks during the years of adol...
This study focuses on the difficulties to maintain a weight loss and behavioural change after having been in a institutional intermittent weight loss programme. We explore if including a ...
This study will test the effectiveness of an intervention for Veterans diagnosed with Alzheimer's Disease (AD) or Traumatic Brain Injury (TBI) and the burden on their informal (family/frie...
Nowadays pregnant women are treated with acetylsalicylic acid (ASA) if they are diagnosed to high-risk of pre-eclampsia (PE) meaning a total of 10% of the total PE screened patient populat...
The CATIE Alzheimer's Disease Trial is part of the Clinical Antipsychotic Trials of Intervention Effectiveness (CATIE) Project. The study is for people with Alzheimer's disease who are ha...
Not an aneurysm but a well-defined collection of blood and CONNECTIVE TISSUE outside the wall of a blood vessel or the heart. It is the containment of a ruptured blood vessel or heart, such as sealing a rupture of the left ventricle. False aneurysm is formed by organized THROMBUS and HEMATOMA in surrounding tissue.
Tissue NECROSIS in any area of the brain, including the CEREBRAL HEMISPHERES, the CEREBELLUM, and the BRAIN STEM. Brain infarction is the result of a cascade of events initiated by inadequate blood flow through the brain that is followed by HYPOXIA and HYPOGLYCEMIA in brain tissue. Damage may be temporary, permanent, selective or pan-necrosis.
Localized reduction of blood flow to brain tissue due to arterial obstruction or systemic hypoperfusion. This frequently occurs in conjunction with brain hypoxia (HYPOXIA, BRAIN). Prolonged ischemia is associated with BRAIN INFARCTION.
Bleeding within the brain as a result of penetrating and nonpenetrating CRANIOCEREBRAL TRAUMA. Traumatically induced hemorrhages may occur in any area of the brain, including the CEREBRUM; BRAIN STEM (see BRAIN STEM HEMORRHAGE, TRAUMATIC); and CEREBELLUM.
A condition characterized by long-standing brain dysfunction or damage, usually of three months duration or longer. Potential etiologies include BRAIN INFARCTION; certain NEURODEGENERATIVE DISORDERS; CRANIOCEREBRAL TRAUMA; ANOXIA, BRAIN; ENCEPHALITIS; certain NEUROTOXICITY SYNDROMES; metabolic disorders (see BRAIN DISEASES, METABOLIC); and other conditions.