Monitoring of Thyroid Malfunction and Therapies in Pregnancy and the Postpartum Period: A Systematic Up-Dated Critical Review of the Literature.

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Summary of "Monitoring of Thyroid Malfunction and Therapies in Pregnancy and the Postpartum Period: A Systematic Up-Dated Critical Review of the Literature."

From the very beginning of pregnancy, the maternal thyroid has to adapt to increased thyroid hormone secretion of up to 50 %. This is paralleled by changes in thyroid-stimulating-hormone (TSH) secretion and by the thyroid-topic action of human chorionic gonadotropin (hCG). Thus, hypothyroidism as well as hyperthyroidism may occur. Many women exhibit pre-existing thyroid diseases. This review tries to add the most recently published approaches to diagnosing thyroid malfunction in pregnancy to existing guidelines.


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This article was published in the following journal.

Name: Therapeutic drug monitoring
ISSN: 1536-3694


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Medical and Biotech [MESH] Definitions

Transient autoimmune thyroiditis occurring in the POSTPARTUM PERIOD. It is characterized by the presence of high titers of AUTOANTIBODIES against THYROID PEROXIDASE and THYROGLOBULIN. Clinical signs include the triphasic thyroid hormone pattern: beginning with THYROTOXICOSIS, followed with HYPOTHYROIDISM, then return to euthyroid state by 1 year postpartum.

The health of women during PREGNANCY; childbirth and the POSTPARTUM PERIOD.

In females, the period that is shortly after giving birth (PARTURITION).

A term used to describe pregnancies that exceed the upper limit of a normal gestational period. In humans, a prolonged pregnancy is defined as one that extends beyond 42 weeks (294 days) after the first day of the last menstrual period (MENSTRUATION), or birth with gestational age of 41 weeks or more.

The beginning third of a human PREGNANCY, from the first day of the last normal menstrual period (MENSTRUATION) through the completion of 14 weeks (98 days) of gestation.

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