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We propose a theory for the drying of liquid droplets of surfactant solutions. We show that the added surfactant hinders droplet receding and facilitates the droplet spreading, causing complex behavior of the contact line of an evaporating droplet: the contact line first recedes and then advances and finally recedes again. We also show that surfactant can change the deposition pattern from mountain-like to volcano-like and then to coffee-ring-like. Specially, when the contact line motion undergoes a clear receding-advancing transition, a two-ring pattern is formed. The mechanism of the two-ring formation is different from the stick-slip mechanism proposed previously, and may be tested experimentally.
This article was published in the following journal.
Name: Langmuir : the ACS journal of surfaces and colloids
Processes for separating oil-water mixtures are critical to operations in energy and water. However, existing separation methods pose efficiency limitations, as well as environmental and safety challe...
This review presents experimental studies of phenomena occurring in droplets of various liquids under the effect of nanosecond spark discharges. Inorganic liquids and liquids of biological origin are ...
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Inkjet printing is here employed for the first time as a method to produce femtoliter (fL) scale oil droplets dispersed in water. In particular, picoliter (pL) scale fluorinated oil (FC40) droplets ar...
Microwave vacuum drying is an innovative technology for drying fruits and vegetables. However, this technology has not been well explored for drying seafood. In the present study, effects of microwave...
The purpose of this study is to compare the changes in blood lipids and feelings of satiety after consumption of acid stable or acid unstable oil-in-water emulsions in which the droplets a...
The purpose of this study is to compare the changes in blood lipids and feelings of satiety after consumption of oil-in-water emulsions in which the droplets are in either the liquid or so...
The present study was designed to evaluate, in premature babies with RDS breathing spontaneously, the efficacy of combined treatment with nasal continuous positive airway pressure (CPAP) a...
The objective of this pilot study is to examine the feasibility and safety of performing a larger trial to assess outcomes following treatment of meconium aspiration syndrome with surfacta...
Inherited deficiencies in any one of 3 genes (surfactant protein B, surfactant protein C, and ATP-binding cassette transporter A3) can cause neonatal respiratory distress syndrome by disru...
A pulmonary surfactant associated protein that plays a role in alveolar stability by lowering the surface tension at the air-liquid interface. It is a membrane-bound protein that constitutes 1-2% of the pulmonary surfactant mass. Pulmonary surfactant-associated protein C is one of the most hydrophobic peptides yet isolated and contains an alpha-helical domain with a central poly-valine segment that binds to phospholipid bilayers.
A pulmonary surfactant associated-protein that plays an essential role in alveolar stability by lowering the surface tension at the air-liquid interface. Inherited deficiency of pulmonary surfactant-associated protein B is one cause of RESPIRATORY DISTRESS SYNDROME, NEWBORN.
An abundant pulmonary surfactant-associated protein that binds to a variety of lung pathogens and enhances their opsinization and killing by phagocytic cells. Surfactant protein D contains a N-terminal collagen-like domain and a C-terminal lectin domain that are characteristic of members of the collectin family of proteins.
An abundant pulmonary surfactant-associated protein that binds to a variety of lung pathogens, resulting in their opsinization. It also stimulates MACROPHAGES to undergo PHAGOCYTOSIS of microorganisms. Surfactant protein A contains a N-terminal collagen-like domain and a C-terminal lectin domain that are characteristic of members of the collectin family of proteins.
A perilipin protein characterized by an extensive 11-mer repeat region, which forms five adjacent alpha-helices. It is expressed primarily in WHITE ADIPOSE TISSUE and differentiating ADIPOCYTES, as well as skeletal muscle and heart. It is soluble in the cytoplasm but re-localizes to the surface of LIPID DROPLETS under high lipid conditions.