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There is emerging evidence that hydrogen-rich water (H2-water) has beneficial effects on the physiological responses to exercise. However, few studies investigate its ergogenic potential. This randomized, controlled trial examined the effects of H2-water ingestion on physiological responses and exercise performance during incremental treadmill running. In a double-blind crossover design, fourteen endurance-trained male runners (34±4 years old; 63.1±7.2 kg; 1.72±0.05 m) were randomly assigned to ingest two doses of 290-mL H2-water or placebo on each occasion. The first bolus was given before six four-minute submaximal running bouts, and the second bolus was consumed before the maximal incremental running test. Expired gas, heart rate (HR), and ratings of perceived exertion (RPE) were recorded; blood samples were collected at the end of each submaximal stage and post maximal running test. Cardiorespiratory responses, RPE, and blood gas indices were not significantly different at each submaximal running intensity (range: 34-91%V̇O2max) between H2-water and placebo trials. No statistical difference was observed in running time to exhaustion (618±126 vs. 619±113 s), V̇O2max (56.9±4.4 vs. 57.1±4.7 mL∙kg-1∙min-1), maximal HR (184±7 vs. 184±7 beat∙min-1) and RPE (19±1 vs. 19±1) in the runners between the trials. The results suggest that the ingestion of 290 mL of H2-water before submaximal treadmill running, and an additional dose before the subsequent incremental running to exhaustion was not sufficiently ergogenic in endurance-trained athletes. Novelty bullets: • Acute ingestion of H2-water does not seem to be ergogenic for endurance performance. • A small dose of H2-water does not modulate buffering capacity during intense endurance exercise in athletes.
This article was published in the following journal.
Name: Applied physiology, nutrition, and metabolism = Physiologie appliquee, nutrition et metabolisme
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Chemical compounds which yield hydrogen ions or protons when dissolved in water, whose hydrogen can be replaced by metals or basic radicals, or which react with bases to form salts and water (neutralization). An extension of the term includes substances dissolved in media other than water. (Grant & Hackh's Chemical Dictionary, 5th ed)
A low-energy attractive force between hydrogen and another element. It plays a major role in determining the properties of water, proteins, and other compounds.
Poisoning that results from chronic or acute ingestion, injection, inhalation, or skin absorption of HEAVY METALS. Acute and chronic exposures can cause ANEMIA; KIDNEY and LIVER damage; PULMONARY EDEMA; MEMORY LOSS and behavioral changes; bone deformities in children; and MISCARRIAGE or PREMATURE LABOR in pregnant women.
A sodium-hydrogen antiporter expressed primarily by EPITHELIAL CELLS in the kidneys, it localizes to the apical membrane of the PROXIMAL KIDNEY TUBULE, where it functions in sodium and water reabsorption and possibly calcium homeostasis. It also is expressed in heart, brain, and lung tissues and is resistant to AMILORIDE inhibition.
Disorders associated with acute or chronic exposure to compounds containing ARSENIC (ARSENICALS) which may be fatal. Acute oral ingestion is associated with gastrointestinal symptoms and an encephalopathy which may manifest as SEIZURES, mental status changes, and COMA. Chronic exposure is associated with mucosal irritation, desquamating rash, myalgias, peripheral neuropathy, and white transverse (Mees) lines in the fingernails. (Adams et al., Principles of Neurology, 6th ed, p1212)
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