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Autologous Stem Cell Rescue for Primary Amyloidosis

2014-08-27 03:50:15 | BioPortfolio

Summary

To evaluate the role of high dose therapy and autologous hematopoietic cell transplant for amyloidosis.

Description

To learn about the use of high dose chemotherapy followed by transplantation using peripheral blood stem cells.

Study Design

Allocation: Non-Randomized, Control: Uncontrolled, Endpoint Classification: Safety/Efficacy Study, Intervention Model: Parallel Assignment, Masking: Open Label, Primary Purpose: Treatment

Conditions

Amyloidosis

Intervention

high dose chemo then auto hematopoietic cell transplant

Location

Stanford University School of Medicine
Stanford
California
United States
94305

Status

Active, not recruiting

Source

Stanford University

Results (where available)

View Results

Links

Published on BioPortfolio: 2014-08-27T03:50:15-0400

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PubMed Articles [33240 Associated PubMed Articles listed on BioPortfolio]

A comparative study of induction chemotherapy with or without autologous hematopoietic stem cell transplantation in the treatment of newly diagnosed young medium/high risk diffuse large B cell lymphoma patients.

To compare the efficacy of induction chemotherapy with or without autologous hematopoietic stem cell transplantation (auto-HSCT) for newly diagnosed young diffuse large B cell lymphoma (DLBCL) patient...

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Medical and Biotech [MESH] Definitions

Clonal hematopoietic stem cell disorders characterized by dysplasia in one or more hematopoietic cell lineages. They predominantly affect patients over 60, are considered preleukemic conditions, and have high probability of transformation into ACUTE MYELOID LEUKEMIA.

A specific immune response elicited by a specific dose of an immunologically active substance or cell in an organism, tissue, or cell.

The dose amount of poisonous or toxic substance or dose of ionizing radiation required to kill 50% of the tested population.

Glycoproteins with a wide distribution on hematopoietic and non-hematopoietic cells and strongly expressed on macrophages. CD58 mediates cell adhesion by binding to CD2; (ANTIGENS, CD2); and this enhances antigen-specific T-cell activation.

Glycoproteins with a wide distribution on hematopoietic and non-hematopoietic cells and strongly expressed on MACROPHAGES. CD58 mediates cell adhesion by binding to CD2; (CD2 ANTIGENS); and this enhances antigen-specific T-cell activation.

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