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This study investigates the role of two endogenous inhibitory mechanisms; exercise-induced hypoalgesia (EIH) and a conditioning painful stimulus (CPM) on passive joint range of motion, passive resistive torque and pain sensitivity. The study is a randomized, repeated-measures cross-over study.
The increase in range of motion following stretching is related to changes in the tolerance to stretch. The mechanistic relationships behind these changes in subject sensation are however unknown but could be related to endogenous modulation of pain.
Exercise induced hypoalgesia (EIH), Static stretch (SS), Rest, Conditioned pain modulation (CPM)
University College of Northern Denmark
University College of Northern Denmark
Published on BioPortfolio: 2019-03-28T08:27:23-0400
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