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Endogenous Modulation of Pain and Stretch Tolerance

2019-03-28 08:27:23 | BioPortfolio

Summary

This study investigates the role of two endogenous inhibitory mechanisms; exercise-induced hypoalgesia (EIH) and a conditioning painful stimulus (CPM) on passive joint range of motion, passive resistive torque and pain sensitivity. The study is a randomized, repeated-measures cross-over study.

Description

The increase in range of motion following stretching is related to changes in the tolerance to stretch. The mechanistic relationships behind these changes in subject sensation are however unknown but could be related to endogenous modulation of pain.

Study Design

Conditions

Pain Threshold

Intervention

Exercise induced hypoalgesia (EIH), Static stretch (SS), Rest, Conditioned pain modulation (CPM)

Location

University College of Northern Denmark
Aalborg
Denmark
9220

Status

Completed

Source

University College of Northern Denmark

Results (where available)

View Results

Links

Published on BioPortfolio: 2019-03-28T08:27:23-0400

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A symptom complex characterized by pain and weakness in SKELETAL MUSCLE group associated with exercise, such as leg pain and weakness brought on by walking. Such muscle limpness disappears after a brief rest and is often relates to arterial STENOSIS; muscle ISCHEMIA; and accumulation of LACTATE.

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