Endocrine Dysfunction and Quality of Life After Subarachnoid Hemorrhage
Summary
Investigation of the incidence of endocrine dysfunction following subarachnoid hemorrhage (SAH), and to see if this has a relation to CNS lesions as evaluated by MRI and to common symptoms after SAH such as general exhaustion, lack of initiative, increased sleep demand and reduced quality of life.
Study Design
Observational Model: Cohort, Time Perspective: Prospective
Conditions
Endocrine Dysfunction
Location
Department of Neurosurgery
Lund
Sweden
Status
Recruiting
Source
Lund University Hospital
Results (where available)
Links
- Source: http://clinicaltrials.gov/show/NCT01101711
- Information obtained from ClinicalTrials.gov on July 15, 2010
Published on BioPortfolio: 2014-08-27T03:14:39-0400
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The purpose of this study is to evaluate radiotherapy followed by endocrine therapy in comparison with endocrine therapy alone for PSA failure after radical prostatectomy.
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Medical and Biotech [MESH] Definitions
Diagnostic Techniques, Endocrine
Methods and procedures for the diagnosis of diseases or dysfunction of the endocrine glands or demonstration of their physiological processes.
Endocrine Disruptors
Exogenous agents, synthetic and naturally occurring, which are capable of disrupting the functions of the ENDOCRINE SYSTEM including the maintenance of HOMEOSTASIS and the regulation of developmental processes. Endocrine disruptors are compounds that can mimic HORMONES, or enhance or block the binding of hormones to their receptors, or otherwise lead to activating or inhibiting the endocrine signaling pathways and hormone metabolism.
Endocrine Gland Neoplasms
Tumors or cancer of the ENDOCRINE GLANDS.
Endocrine Surgical Procedures
Surgery performed on any endocrine gland.
Multiple Endocrine Neoplasia
A group of autosomal dominant diseases characterized by the combined occurrence of tumors involving two or more ENDOCRINE GLANDS that secrete PEPTIDE HORMONES or AMINES. These neoplasias are often benign but can be malignant. They are classified by the endocrine glands involved and the degree of aggressiveness. The two major forms are MEN1 and MEN2 with gene mutations on CHROMOSOME 11 and CHROMOSOME 10, respectively.
